FRCS (ORL-HNS) · SECTION 1 SBA

Otogenic sigmoid sinus thrombosis

A 17-year-old with acute mastoiditis remains septic with headache and papilloedema. Contrast CT shows an empty-delta appearance in the sigmoid sinus and a perisinus abscess. What is the best management principle?

CHOOSE ONE ANSWER

Best of five

  1. A. Anticoagulate and defer source control
  2. B. Urgent IV antibiotics and mastoid source control with multidisciplinary discussion of anticoagulation
  3. C. Needle aspirate the jugular bulb through the neck
  4. D. Observe because venous sinuses often recanalise spontaneously
  5. E. Perform lumbar puncture before antibiotics
ANSWER AND REASONING

B. Urgent IV antibiotics and mastoid source control with multidisciplinary discussion of anticoagulation

This is complicated otomastoiditis with septic lateral-sinus thrombosis: resuscitation, IV antibiotics, source control and neurosurgical/infectious-disease input take priority.

Why every option is right or wrong

A. Anticoagulate and defer source control: Anticoagulation is individualised and cannot substitute for eradication of the otogenic focus.

B. Urgent IV antibiotics and mastoid source control with multidisciplinary discussion of anticoagulation: Sepsis, intracranial venous thrombosis and perisinus pus require broad antimicrobial treatment and operative control of mastoid disease; anticoagulation depends on propagation, bleeding risk and specialist consensus.

C. Needle aspirate the jugular bulb through the neck: This is hazardous and does not control the mastoid/perisinus infection.

D. Observe because venous sinuses often recanalise spontaneously: Untreated septic thrombosis risks propagation, meningitis, embolic complications and raised intracranial pressure.

E. Perform lumbar puncture before antibiotics: Papilloedema raises concern for intracranial hypertension; lumbar puncture before imaging-based safety assessment is inappropriate.

What if the scenario changed?

If imaging showed only a non-occlusive filling defect without mastoid infection or sepsis, the balance would shift toward a primary cerebral-venous-thrombosis pathway.

EDUCATIONAL USE

Independent Clinora educational preparation material; not official JCIE content, an accredited programme, or a substitute for local policy and specialist clinical judgement.

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